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We have further shown that this effect could originate from the hypothalamus, as a dual agonist of GLP-1RA with GIP had a synergetic effect in decreasing feeding behavior compared to GLP-1 or GIP alone when delivered to the hypothalamus in an ICV manner [83]

Specifically, the relative risks were reduced by approximately 37% for Alzheimers disease, 41% for Lewy body dementia, and 56% for vascular dementia

While the GLP-1 is the catalyst, the fatigue itself is often magnified by other factors that can be easily overlooked

The GLP-1 benefits become a secondary advantage of a healthier gut ecosystem

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