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Exendin-4 inhibits the remodeling in the remote myocardium of rats following acute MI by attenuating -catenin activation and activating glycogen synthase kinase-3, -arrestin-2 and protein phosphatase 2A [12]

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We also reveal that GLP-1(2836) activated sAC by increasing production of its substrate ATP, through enhanced glucose metabolism and reduced FAO by direct inhibitory interactions with MTP
