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According to a recent Bloomberg article, industry investors have put the current black market for these peptides somewhere between $1 and $3 billion

However, these studies lack supporting human clinical data, and the specific molecular mechanisms underlying bile acid metabolism regulation remain to be validated

For more on this, see our guide to How to Store Peptides: Storage and Shelf Life Guide, and how it is evaluated and treated at Perfect B in Doral

The trade-off is cost

Additionally, analogues containing the quinolinium scaffold lacked inhibitory activity against enzymes in the NAD + salvage pathway that bind nicotinamide-containing substrate, including NAMPT[27, 32] and the NAD + -dependent SIRT1 enzyme, which deacetylates NAD + to produce NA, a product inhibitor of SIRT1.[33] These results suggest that quinolinium-based NNMT inhibitors achieve selectivity by specifically interacting with the NA-binding pocket of NNMT,[17] unlike several known non-selective methyltransferase inhibitors that interact with the SAM-binding pocket, which is highly conserved among SAM-dependent methyltransferases.[34, 35] Membrane-permeable NNMT inhibitors reduced intracellular 1-MNA levels in a concentration-dependent manner and at pharmacologically relevant concentrations that did not impact cell viability, suggesting these small molecules interact directly with NNMT in cells

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