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The distal upstream region of insulin-like growth factor-binding protein-1 enhances its expression in endometrial stromal cells during decidualization

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To further explore the potential mechanism of MIA3 in HCC cells, we constructed overexpressed HCC cells for RNA-seq experiments, and the analysis of RNA-seq results showed that compared with HCC cells in the control group, the molecular functions and signalling pathways of cells overexpressing MIA3 were significantly activated in protein binding and endoplasmic reticulum protein processing
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