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The transitional cycle of asymmetrical, symmetry-approaching and pseudo-symmetrical conformations during heme transport is unique to CydDC and highlights the mechanistic possibilities for the specialized substrate transport facilitated by heterodimeric ABC transporters and their diverse conformational space (Supplementary Fig

Studies have shown that excessive HOCl/OH generation during neutrophil activation induces extensive DNA damage and subsequent DNA repair mechanisms which will result in NETosis [246, 247]