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Thus, the potency of KRAS G12C inhibition is reduced by increased RTK activity, that promotes cycling of KRAS G12C to its active GTP-bound form, hindering KRAS G12C drug inhibition (94)

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Curr Med Chem 2003;10:13891398

The clinical pharmacologists/scientists should be aware of the literature, as well as in vitro and preclinical pharmacology models to determine if a suitable candidate biomarker should be included in an FIH study