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Induction of apoptosis by cancer chemotherapy

Increased generation of reactive oxygen species, decreased activity of antioxidant systems or both mechanisms may be involved in the occurrence of oxidative stress ( A study showed that lenalidomide attenuates oxidative cardiovascular tissue damage and apoptosis in obese mice by inhibiting tumor necrosis factor (Zhu et al., 2014)

To this end, we generated reconstituted cell line expressing WT HK2 or a truncation mutant of HK2 lacking the N-terminal 15 amino acids (N15), which is essential for its association with mitochondria outer membrane 51

Thus, their brain pharmacokinetics are altered by P-gp and response to treatment is affected (Lin et al., 2011)

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The glitazars paradox: cardiotoxicity of the metabolically beneficial dual PPAR and PPAR activation