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Inflammatory endothelial cells and macrophages overexpress VCAM-1/IL-1R, which enables dual targeting via VCAM-1-binding NPs that restore TIMP3 and IL-1R-antagonist systems that promote M2 polarization through rapamycin, resulting in synergistic plaque stabilization and immunomodulation [243,244,245]
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Resistance training is non-negotiable, along with proper protein intake
The oncometabolite fumarate promotes pseudohypoxia through noncanonical activation of nf-Kb signaling
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