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First, we conducted the cytotoxicity experiments of Fe 1 /NC SACs

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Mechanisms by which metabolites of DCVC and other nephrotoxic haloalkene cysteine S -conjugates alter thiol status may involve direct inhibition of glutathione reductase (Lock and Schnellmann, 1990) and generation of oxidative stress that leads indirectly to oxidation of thiol compounds (Chen et al., 1990)