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It is reasonable to assume that, in C57BL/6 mice, SIRT1 overexpression alleviates age-related cochlear hair cell loss and hearing loss through activation of the PINK1-Parkin pathway to ease the cellular survival burden due to oxidative stress

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doi:10.1007/s00330-020-06696-x Gutmann C, Siow R, Gwozdz AM, Saha P, Smith A

The hypothesis of the gain of function in GCase theorizes that the mutant form of protein is recognized as misfolded, thus placing stress on mechanisms of protein degradation and interfering with -synuclein breakdown and consequent endoplasmic reticulum stress, as observed in autophagy perturbations in cells derived from patients with GBA1 -associated PD ( 2.5.2 Clinical studies Monogenic PD cases due to GBA1 loss of function and subsequent substrate accumulation have been investigated in cellular and animal models and in studies of human brains of patients with GBA1 -associated PD showing to particularly affect pars compacta of SN, putamen, cerebellum, and amygdala and frontal cortex (Moors et al., 2019)

Effects of hyperbaric oxygen on T helper 17/regulatory T polarization in antigen and collagen-induced arthritis: Hypoxia-inducible factor-1 as a target

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