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In controlled laboratory environments, GHK-CU is primarily used in in-vitro and experimental research models to investigate peptidereceptor interactions, copper-dependent signaling mechanisms, and intracellular communication processes

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The main mechanism responsible for Graves hyperthyroidism is the activation of the thyroid-stimulating hormone receptor (TSHR) by autoantibodies that act as agonists, causing thyrocyte proliferation and hyperfunction

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