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This overall increased oxidative burden may contribute to chronic cellular stress, mitochondrial injury, apoptosis, and may induce tubular cell injury [347, 348]
Redox-implications associated with the formation of complexes between copper ions and reduced or oxidized glutathione
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Studies have associated the disease with increased levels of IL-6 and TNF-alpha [34]
Yang Z, Attygalle AB
Regional distribution of the cells expressing glycine receptor beta subunit mRNA in the rat brain
