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The valproic acid animal model of ASD illustrates E/I imbalances caused by the differentiation of glutamatergic neurons and decreased numbers of GABA-ergic neurons [26]

doi: 10.1186/1472-6874-7-5

We want to thank Dr Laura Tronci (CF group), Dr Pablo Oriol Valls and Dr Andrew Trinh (AE group) for performing experiments in relation to the study of KRAS mutant cell lines that have not been included in this work

The uniform upregulation of important metabolites underscores a potential compensatory adaptation by the bacterium to maintain viability under the challenges of altered metabolic and oxidative states

Notably, PD has exhibited the faster growth among neurological disorders in terms of both DALYs and mortality over recent decades [37]

doi: 10.1136/bjophthalmol-2020-316245 111