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Elevated intracellular iron levels disrupt the IRP/IRE signaling pathway, leading to increased expression of APP (35,36) and A production

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This implies that GST polymorphism and its tissue specific differential effects are important determinants to correlate cancer risk and ITC intake

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Notably, the knockout of the HDAC3 gene or the use of HDAC3 inhibitors can effectively alleviate the inhibition of GPX4, ferroptosis, and renal function damage associated with fibrosis (Zhang L