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Further study of this model and this subgroup of ASD patients should improve our understanding of the pathophysiology and potential risk factors that lead to the metabolic, brain and behavior abnormalities associated with ASD

Although CPT1A and CPT1B exhibit considerable sequence similarity, the sensitivity of these two enzymes to their physiological inhibitor, malonyl-CoA, differs greatly (CPT1A has a ten-fold higher Ki for malonyl-CoA) 39,40

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