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CONCLUSIONS Development of novel therapeutics that restore the tight junction barrier after gliadin exposure in patients with celiac disease is crucial, given that the only current treatment option is adherence to a strict, gluten-free diet, which is not wholly successful in all patients with celiac

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A high-fat diet impairs liver regeneration in C57BL/6 mice through overexpression of the NF-kappaB inhibitor, IkappaBalpha