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Conclusion This study reveals significant metabolic alterations in congenital hypothyroid patients, including reduced long-chain acylcarnitine levels and inferred low CPT-I activity, which are suggestive of impaired -oxidation and energy metabolism

An incidence for these reactions is difficult to estimate due to the confounding effects of the underlying pathology

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Contradictory results in favor or against the pro-inflammatory effect of these products consumption in humans were published [151]

The primary functions of FC and acylcarnitines (AC), mainly acetyl-L-carnitine (ALC) in humans, are the transport of long-chain fatty acids (FAs) to the mitochondrial matrix and maintaining the mitochondrial homeostasis of coenzyme A (CoA) during mitochondrial oxidation of these acids [49]

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