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Glycation-altered proteolysis as a pathobiologic mechanism that links dietary glycemic index, aging, and age-related disease (in non diabetics)

Therapeutic targeting of proteostasis in metabolic diseases Targeting HSPs in metabolic diseases Given their central roles in metabolic homeostasis, heat shock proteins (HSPs) have emerged as attractive therapeutic targets in metabolic disease

[8] In the kidney , gluconeogenesis takes place in the proximal tubule cells

When MAPK/ERK1/2 and p38 MAPK signaling pathways begin, the result is the common denominator of signaling pathways, which alter gene expression, metabolism, and cell proliferation [43]

This result was associated with the expression of myogenic factors and autophagy markers, in addition to a decrease in endoplasmic reticulum stress [80, 81]

A previous study indicated that NRF2 is an important and novel transcriptional regulator of ferroptosis in hepatocellular carcinoma (HCC) cells, and NRF2 activation can inhibit ferroptosis in HCC cells [67,68,69]