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One mouse was euthanized on day 12 in MK-801 group due to sickness

Pharmacologic strategies to target mitochondrial dysfunction Mitochondrial stress pathways have been implicated in disease manifestations of mitochondrial dysfunction and could highlight promising therapeutic targets [164-166]

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Furthermore, reversible oxidative post-translational modifications of protein cysteines, particularly GS-ylation, were dramatically increased in the aortas of a Marfan syndrome mouse model