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In addition, GSH has been shown to regulate nitric oxide homeostasis (Hogg, 2002), modulate the activity of proteins by post-translational modification (protein S-glutathionylation) (Pompella et al., 2003), and modulate the activity of neurotransmitter receptors (Oja et al., 2000)

It sustains adequate redox cell signaling since a physiologic level of oxidative stress is fundamental for controlling life processes via redox signaling, but excessive oxidation causes cell and tissue damage

2016;11:e0156268

In conclusion, we developed a semi-mechanistic popPK model to investigate the PK variability of IV busulfan in pediatric HCT recipients

IL-1 Induces pathologically activated osteoclasts bearing extremely high levels of resorbing activity: A possible pathological subpopulation of osteoclasts, accompanied by suppressed expression of kindlin-3 and talin-1

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