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Recently, manganese-based superoxide dismutase like synthetic AO synzyme (M40403), and azymes[9] have also been added to synthetic AOs group of N-acetylcysteine, pencillinamine and tetracyclines.[4] Oxidative stress (OS) leads to molecular damage resulting from disturbances in redox signaling and control due to an imbalance between oxidants and AOs oxidants ROS.[10,11,12] OS is considered to play an important role in the pathogenesis of chronic inflammatory conditions like non-insulin dependent diabetes mellitus, cardiovascular disease, metabolic syndrome,[11,13,14,15] acute respiratory distress syndrome,[5,16] and periodontal disease.[5,17,18,19,20,21,22] Recently, it has been proposed as a central link between systemic diseases and periodontitis.[23] Ubiquitous AO master, glutathione is a low-molecular weight, major non-protein cellular thiol, present in both eukaryotic as well as prokaryotic cells,[24,25,26] and exist in every single cell of the human body.[24] It is a potent AO that prevents ROS mediated damage to essential cellular components and acts as a cofactor for enzymes in the destruction of ROS

These neurons produce broad, sluggish action potentials that promote slow rhythmic activity and boost calcium influx

These protective effects were attributed to antioxidant mechanisms that mitigated oxidative stress, as well as the inhibition of key inflammatory mediators, including TGF-, TNF-, and p53

Data on GLY from a previous, unpublished study conducted in cats were used to estimate the variance components for calculating the sample size for the GLY inclusion level study