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The dynamic changes of O can also affect the functional state of hippocampal NSCs and are related to their transition from a resting state to an activated state [291]
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Elucidating non-monotonic dose-response relationships (NMDR) remains essential for determining accurate low-dose EED toxicity thresholds
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Upon activation, proapoptotic proteins form pore complexes on the mitochondrial membrane, resulting in the loss of the mitochondrial membrane potential and the release of apoptotic factors such as cytochrome C, thereby activating the caspase-9/3 cascade [46]
In oncogenesis, NO (at concentrations 12)
