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[DOI] [PubMed] [Google Scholar] 48.Denneny E., Sahota J., Beatson R., Thornton D., Burchell J., Porter J
Nie stanowi jednak swoistego markera diagnostycznego, a jego spadek odzwierciedla zaburzenia rwnowagi redoks i funkcji antyoksydacyjnych komrki

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Individuals with severe kidney or liver disease should approach glutathione therapy with caution and only under direct medical supervision, as these organs play crucial roles in processing the compounds

Furthermore, the relevance of a Grx(SSR) intermediate and an attack by GSH at the glutaredoxin sulfur atom according to monothiol mechanism (i) were postulated for the reduction of the non-glutathione disulfide bond of the HMA4 domain 60

The oxidation of specific tyrosine and methionine residues in apoA-I contributes to its loss of activity (Shao, 2012)
