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Access this article Similar content being viewed by others Data availability The datasets generated during and/or analysed during the current study are available from the corresponding author on reasonable request

Cell-autonomous metabolic deregulation of cancer cells, or metabolic adaptation to microenvironmental cues (lack of nutrients, stromal supply, hypoxia, etc.), represent the triggering event of mitochondria overexploitation to orchestrate nutrient sensing and upload, signaling, and redox circuits

Graphical abstract Keywords: Polyphenols, Apoptosis, Neuronal loss, Beta amyloid, Cognitive dysfunction, Learning and memory impairment, Parkinsonism Introduction Neurodegenerative diseases are a heterogeneous group of chronic, usually age-related disorders that are characterized by the progressive loss of neurons, deformation of the neuron structure, or loss of neuron function [1]

As shown in Figure 2D,E, significantly increased number of apoptotic cells was found in the TBI group relative to the sham group

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Melatonin is a hormone released by the pineal glands in our brain and regulates the sleep-wake up cycle and circadian rhythm