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The primary endpoint was a minimum of 50% reduction in tumor cell count assessed by quantitative multiplex immunofluorescence (qmIF) within the tumor bed of the surgical tissue (post-treatment) compared to the biopsy tissue (pre-treatment)

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The proposed mechanism involves direct interaction between the AEDG sequence and histones H1/3 and H1/6 a form of epigenetic regulation in which the peptide modulates chromatin accessibility at specific gene loci, thereby influencing transcriptional activity without altering the underlying DNA sequence (9)

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APX is located in multiple subcellular compartments, allowing finely tuned scavenging activity