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10eh), while TAP-VK1 analog 3o mainly co-localized in ACE

Low platelet mitochondrial complex I and complex II/III activity in early untreated Parhnsons disease

[168], he proposed that NO may bind to the enzymes of mitochondrial ETC, such as NADH succinate oxidoreductase, NADH ubiquinone oxidoreductase, and cis aconitase and induce uncoupling of OXPHOS followed by the inhibition of glycolysis [66] and depletion of ATP in the cells [169]

Gote V., Bolla P