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Dose-dependent associations existed between L-carnitine levels and risk of coronary artery disease, peripheral artery disease, and CVD but only in participants with concurrently high TMAO levels [3][7][36]
This suggests that L-carnitine enhances tissue-level oxygen utilization, allowing for more efficient recovery from hypoxia-induced stress during resistance training
The triglycerides stored within the LDs are then degraded into FFAs by lysosomal lipases
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doi:10.1046/j.1528-1157.2001.18001
