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Description
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Using novel genetic and chemical techniques to fate map pre-made lung ECM in animal models of lung fibrosis, we uncover a novel mechanism of connective tissue proteolysis at PAJs with subsequent mobilization of pleural macromolecules inwards that feeds and sustains pulmonary fibrosis
The main source of this uncertainty is individual variation in the Widmark r factor , elimination rate, and body composition
Prochazkova D, Bousova I, Wilhelmova N
Crit Care Clin
