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Kelly JW, Colon W, Lai Z, Lashuel HA, McCulloch J, McCutchen SL, Miroy GJ, Peterson SA

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Conclusion: Normal brain GSH in living brain of chronic MA users is consistent with our previous postmortem brain finding and suggests that any oxidative stress caused by MA, at the doses used by our participants, might not be sufficient to cause either a compensatory increase in, or substantial overutilization of, this antioxidant

[DOI] [PMC free article] [PubMed] [Google Scholar] 115.Harnett W

The inhibited SGLT1 activity and reduced expression of GLUT2 during parasitic nematode infection lower enterocyte intracellular glucose (97, 99)

This tight coordination has a critical consequence: copper redox activity is silenced when properly complexed with the tripeptide, preventing the copper from generating reactive oxygen species through the Haber-Weiss reaction pathway