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This results in mitochondrial oxidative stress and dysfunction, ultimately leading to hepatocyte necrosis (Yan et al., 2018)

A Phase II Study of Azacitidine, Venetoclax, and Trametinib in Relapsed or Refractory Acute Myeloid Leukemia Harboring RAS Pathway-Activating Mutations
Similarly, hucMSCs-exosomal miR1792 promotes wound healing by inducing angiogenesis and inhibiting ferroptosis [196]

19 Dysregulated putrescine metabolism may enhance MAOB activity, increasing GABA synthesis and exacerbating tonic inhibition under pathological conditions

The role of polyol pathway in high glucose-induced endothelial cell damages
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