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Rizk Y, Saad N, Arnaout W, Chalah MA, Farah S

[DOI] [PMC free article] [PubMed] [Google Scholar] 181.Muise AM, et al

doi: 10.1016/j.drudis.2014.10.003
While classically it was thought that -arrestins would sterically prevent G proteins from interacting with the receptor, more recently it has been noted that -arrestin and G proteins can bind to the receptor simultaneously, allowing the activation of G protein-mediated signaling [57]

PARP1 inhibition using 3-AB has shown protective effects in various models of ischemia-reperfusion injury
