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Key mechanisms include estrogen dominance with progesterone resistance, immune dysfunction, oxidative stress-induced ferroptosis, and disrupted endometrial receptivity
Epidermal growth factor receptor (EGFR) activation was identified as a basis for the lower response in CRC, with additional EGFR inhibition together with BRAF inhibitor treatment overcoming this issue [7]
With respect to antioxidant interventions, apart from vitamin E, other antioxidants such as carotenoids, ascorbic acid, and flavonoids have been shown to have a very mild effect on preventing oxidative damage (Halliwell 2020
Ravnan et al., 2006
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