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This therapeutic paradox may reflect inadequate patient stratification and failure to distinguish between acute reversible oxidative injury and chronic persistent inflammation

640 Similarly, the age-related increases in skeletal lipoxygenase metabolism appears to influence bone loss, where 9- and 13-HODEs are shown to suppress -catenin mediated Wnt signaling, reducing osteoblast proliferation and promoting apoptosis

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Takakura K

It was established in 1973 that Se is a structural component of the active center of the animal enzyme cellular glutathione peroxidase (GPX1) (6,7)

These free radicals contribute to tiredness, weak immunity, faster ageing rate, and cardiovascular problems