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These results supported that compound 102 was a specific GPX4 activator, and increasing GPX4 activity would be an alternative way to suppress ferroptosis
doi: 10.1159/000481951 198 RenaGHardieDGPearsonER
This selection ensured that only relevant and scientifically robust studies addressing the biological impact of Zn overload were incorporated into the review
2015;10(4):e0124897 33
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