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Potential future applications of PQQ include its use in molecular modeling and docking studies for quantitative drug design based on bioinformatics and structural characterization of the novel drug molecules

revealed an additional transcription start site 233 bp downstream of this promoter, with a 25-fold increase in transcriptional activity [35]

Missing MS/MS acquisition of the IHEVLAPGC * L peptide precursor at m/z 600.8077 was caused by a higher abundant interference at m/z 601.3123, overlapping with the peptide [M+1+2H] 2+ signal

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This resulted in decreased GSH, increased ROS and malondialdehyde, ultimately inhibiting proliferation, clone formation, and inducing ferroptosis in A549 cells