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[86] have discovered that herpes simplex virus type 1 infection can be identified in the brains of patients with AD

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That being said, the findings indicated that there was a dose-dependent relationship between administration of the nootropic and the recorded escape latencies [2]

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[DOI] [PMC free article] [PubMed] [Google Scholar] 62.Setoue M, Ohuchi S, Morita T, Sugiyama K

This finding suggests that -synuclein might facilitate its movement between neurons by interacting with ApoE, which is involved in lipid transport mechanisms (Paslawski et al., 2019)