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Notably, this polymorphism reduced hypoxia-induced GPX3 transcription [197,198]
This pro-angiogenic activity, mediated at least in part through upregulation of VEGF receptor expression, is considered a key contributor to the accelerated tissue repair seen across different injury models
Klooster et al., 2011
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When NF-kB interacts with IkB in the cytoplasm, it is unable to enter the nucleus and bind to the promotor region of proinflammatory genes
