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This study identifies mitochondrial mechanisms contributing to sleep pressure and, thus, puts forward a key role for mitochondria in the broad regulation of brain states and physiological functions

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Two recent papers illustrated that this asymmetry is associated with distribution of metabolic mediators in CD8 + T cells, such that one daughter cell receives more activated mTORC1 and ultimately becomes a terminally differentiated, highly glycolytic Te cell, while the other daughter cell is primed to become a long-lived memory T (Tm) cell (33, 34)