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Zhang Z., Kang S

It is hypothesized that high glucose concentrations in red blood cell (RBC) preservatives lead to increased methylglyoxal (MG) metabolism, causing glycation-driven damage to transfused RBCs

Transgenic mice that develop thyroid-specific autoantigens in a spontaneous manner have, therefore, been very useful and provide a good platform for studying immunological mechanisms of the disease as reported by Merrill and Pandiyan (2018) (Merrill and Pandiyan 2018)

Curr Opin Neurol 23:103110

Chaetocin-induced ROS-mediated apoptosis involves ATM-YAP1 axis and JNK-dependent inhibition of glucose metabolism

949 The excellent ROS elimination ability of PPR NPs gives them potential to inhibit ferroptosis