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In the LPS-induced acutelung injury mouse model, treatment of GHK-Cu also significantly suppressed TNF- and IL-6 expression with a significant decrease of both NF-B p65 and p38 MAPK signaling, suggesting that GHK-Cu is useful for the development of novel anti-inflammatory therapies [22]

Toxicol Appl Pharmacol 204 , 238255, (2005)

It was found that the combined and individual treatment with EGCG and gefitinib inhibited the metastatic ability of CAL-27 cells, which was further accomplished by suppressing the expression level of p-EGFR protein and phosphorylation of ERK, JNK, p38, MMP-2 and AKT proteins [49]

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