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Cyclic GMP is degraded by phosphodiesterases, so if the goal is to increase cGMP, phosphodiesterase (PDE) inhibitors can be used

Taken together, these findings suggest that APR-246 could induce tumor cell death through both reactivation of mutant p53 and inhibition of cellular thiol-dependent redox system proteins (GPX4, TXNDR1, GRX1), providing novel combinatorial strategies for AML therapy

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SK3 channel and mitochondrial ROS mediate NADPH oxidase-independent NETosis induced by calcium influx

10.1186/s12967-023-04179-3 63 GkouviA.TsiogkasS