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e20160668

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doi: 10.1007/BF00965509

Surprisingly, mice with specific genetic deletion of Gpx1 appear phenotypically normal and with normal life span (Ho et al., 1997), suggesting that there are alternative compensatory mechanisms for H 2 O 2 scavenging in Gpx1 deficiency
Epidemiology of Wilson's disease and pathogenic variants of the ATP7B gene leading to diversified protein disfunctions
