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Identification of a novel oleic acid analog with protective effects in multiple cellular models of friedreich ataxia

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Concurrently, peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC-1 ) expression increases 35-fold after 6 weeks of training, driving mitochondrial biogenesis evidenced by increased mitochondrial DNA copy number and respiratory chain complex protein expression ( integrates with the IGF-1/PI3K axis through direct phosphorylation by Akt, creating a feed-forward loop that maintains oxidative capacity specifically when training volumes exceed 150 min/week ( Training reduces myocardial nuclear factor- B (NF- B) activation by 35% and decreases TNF- and IL-6 mRNA expression by 40%50%, effects mediated partly through Akt-dependent inhibition of IKK ( In endothelial cells, training enhances Akt phosphorylation of eNOS at Ser1177, increasing nitric oxide production by 25%35% and improving flow-mediated dilation by 2%4%, adaptations that persist at rest unlike the transient effects of acute exercise (Figure 2)

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