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the ADC of the invention that specifically binds to an Eph receptor is not an ADC of 12G3H11

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Upon drug exposure, cancer cells activate alternative survival pathways including receptor tyrosine kinases (e.g., AXL, IGF-1R), developmental pathways (WNT/-catenin, YAP/TEAD), and stress-response signaling (STAT3, Aurora kinase A), as indicated in recent studies (Wang et al., 2020