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Similarly, proteins and small-molecule antioxidants such as glutathione (GSH) are vulnerable to ROS, leading to impaired cellular redox buffering, mitochondrial dysfunction, and diminished contractile and neuronal function [39]

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To date, however, testing this hypothesis in humans has been complicated by both a relative scarcity of data on postpartum maternal immune function and predominance of infant-centered and binary/categorical measures of breastfeeding behavior (e.g., simple presence/absence of supplementation) 4,5,6

In contrast to classic type 2 diabetes which is a multifactorial (polygenic and environmental contributions) disease, MODY represent a family of monogenic causes of diabetes mellitus
