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doi: 10.1016/j.biortech.2021.126629

doi: 10.5653/cerm.2020.04175 28 PasquarielloRAnipchenkoPPennarossaGCrociatiMZeraniMBreviniTAet al

Homocysteine-induced endothelial dysfunction

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recently showed that direct GPx4 overexpression in the cellular models (A549, H460 and H1299 and H23) of non-small cell lung carcinoma (NSCLC) prevented the radiation-induced lipid peroxidation and, in turn, contributed to radioresistance by blocking the ferroptosis mode of cell death [56]