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Loss of cathepsin L activity promotes claudin-1 overexpression and intestinal neoplasia
The evolution of the concept of stress and the framework of the stress system
Consortium to Establish a Registry for Alzheimer's Disease (CERAD): the first twenty years
J Womens Health (Larchmt) 2010
This tripeptide molecule functions as the primary defense mechanism against oxidative stress, free radical damage, and cellular toxin accumulation
Nasal administration of amyloid-beta peptide decreases cerebral amyloid burden in a mouse model of Alzheimers disease
