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"DIABASI trial." J Endocr Soc 2018

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That is, quinolinic acid may have toxic effects on the CNS, contribute to oxidative stress and neuroinflammation in both dopaminergic and glutaminergic neurons (198)

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doi: 10.1007/bf00755687

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More importantly, pharmacological GCL inhibition impaired lymphomagenesis in a mouse model of B-cell lymphoma

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DOI:10.1042/BJ20021684 37

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Interestingly, Lepr db/db macrophages secreted significantly more IL-1 in a steady-state, as well as upon stimulation with lipopolysaccharide (LPS) and adenosine triphosphate (ATP) (Fig

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