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[DOI] [PubMed] [Google Scholar] 109.Pan S., Li J., Zhang X., Li Y., Yan J., Zhang W., Hu S

The mechanisms reported were: (i) prevention of the downregulation of Bcl-X L

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Exogenous supplementation of propionate in GPR41 / mice resulted in increased blood pressure, whereas the same treatment in Olfr78 / mice resulted in a significant decrease in blood pressure, suggesting that the activation of GPR41 and Olfr78 by SCFA can have antagonistic effects and is involved in the regulation of blood pressure [95, 96]